池田 弘子 薬物治療学教室 職 位 准教授 博士(歯学) 星薬科大学大学院

Hoshi University
氏
名
池田
所
属
薬物治療学教室
学
位
博士(歯学)
最終学歴
教育活動
(担当授業科目)
弘子
職
位
准教授
星薬科大学大学院薬学研究科博士前期課程
疾患と薬物治療ⅡB
テーラーメイド薬物治療
薬物治療演習
薬学英語ⅡA
研究活動
(研究テーマ)
中枢によるエネルギー代謝調節機構の解明
肥満に伴う中枢および末梢神経機能の変化とその発現機構の解明
中枢神経系―ドパミン神経機能の神経化学的・行動薬理学的研究
所属学会
日本薬学会、日本薬理学会、日本神経精神薬理学会、
日本神経科学会、日本肥満学会
Society for Neuroscience、American Diabetes Association
学内活動
(役職・委員等)
学生部長補佐(学生部委員会副委員長)、学寮員会副委員長
新コアカリキュラム検討委員会副委員長、施設利用検討委員会委員長、
将来計画検討委員会委員、研究総合戦略検討委員会委員、FD 実施検討委員
会委員、能動的学修支援システム対応委員会委員、大学院昼夜開講運営委員
会委員、実習委員会委員、ハラスメント相談員
星薬科大学生協共同組合理事、揺籃会理事
学外活動
(委員会委員等)
日本薬理学会評議員、日本薬理学会代議員
厚生労働省薬剤師試験委員
研究業績等
(主な著書・論文)
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IKEDA, H., ADACHI, K., FUJITA, S., TOMIYAMA, K., SAIGUSA, T., KOBAYASHI, M., KOSHIKAWA,
N., WADDINGTON, J.L.: Investigating complex basal ganglia circuitry in the regu lation of motor
behaviour, with particular focus on orofacial movement. Behav. Pharmacol., 26, 18-32 (2015)
TANAKA, K., NAKANISHI, Y., SEKINO, S., IKEGAMI, M., IKEDA, H., KAMEI, J.: Fentanyl produces
an anti-hyperalgesic effect through the suppression of sodium channels in mice with painful diabetic
neuropathy. Eur. J. Pharmacol., 733, 68-74 (2014)
SHIMIZU, T., SUGIHARA, E., YAMAGUCHI-IWAI, S., TAMAKI, S., KOYAMA, Y., KAMEL, W.,
UEKI, A., ISHIKAWA, T., CHIYODA, T., OSUKA, S., ONISHI, N., IKEDA, H., KAMEI, J., MATSUO,
K., FUKUCHI, Y., NAGAI, T., TOGUCHIDA, J., TOYAMA, Y., MUTO, A., SAYA, H.: IGF2 preserves
osteosarcoma cell survival by creating an autophagic state of dormancy that protects cells against
chemotherapeutic stress. Cancer Res., 74, 6531-6541 (2014)
IKEDA, H., SAIGUSA, T., KAMEI, J., KOSHIKAWA, N., COOLS, A.R.: Spiraling dopaminergic circuitry
from the ventral striatum to dorsal striatum is an effective feed -forward loop. Neuroscience, 241, 126-134
(2013)
IKEDA, H., IKEGAMI, M., KAI, M., OHSAWA, M., KAMEI, J.: Activation of spinal cannabinoid CB 2
receptors inhibits neuropathic pain in streptozotocin -induced diabetic mice. Neuroscience, 250, 446 -454
(2013)
IKEDA, H., KOSHIKAWA, N., COOLS, A.R: Accumbal core: Essential link in feed -forward spiraling
striato-nigro-striatal in series connected loop. Neuroscience 252, 60 -67 (2013)
IKEGAMI, M., IKEDA, H., ISHIKAWA, Y., OHSAWA, M., OHASHI, T., KAI, M., KAMEI, A., KMEI, J.:
Olanzapine induces glucose intolerance through the activation of AMPK in the mouse hypothalamus. Eur.
J. Pharmacol., 718, 376-382 (2013)
IKEGAMI, M., IKEDA, H., OHASHI, T., OHSAWA, M., ISHIKAWA, Y., KAI, M., KAMEI, A., KAMEI,
J.: Olanzapine increases hepatic glucose production through the activation of hypothalamic AMPK.
Diabetes Obes. Metab., 15, 1158-1135 (2013)
IKEGAMI, M., IKEDA, H., OHASHI, T., KAI, M., OSADA, M., KAMEI, A., KAMEI, J.:
Olanzapine-induced hyperglycemia: Possible involvement of histaminergic, dopaminergic and adrenergic
functions in the central nervous system. Neuroe ndocrinology, 98, 224-232 (2013)
KAMEI, J., NAKANISHI, Y., ASATO, M., IKEDA, H.: Fentanyl enhances the excitability of rapidly
adapting recetpors to cause cough via the enhancement of histamine release in the airways. Cough, 9, 3
(2013)
IKEDA, H., KAMEI, J., KOSHIKAWA, N., COOLS, A.R.: Nucleus accumbens and dopamine -mediated
turning behavior of the rat: role of accumbal non-dopaminergic receptors. J. Pharmacol. Sci., 120, 152-164
(2012)
IKEDA, H., MIYATAKE, M., KOSHIKAWA, N., OCHIAI, K., YAMADA, K., KISS, A ., DONLIN, M.J.,
PANNETON, W.M., CHURCHILL, J.D., GREEN, M., SIDDIQUI, A.M., LEINWEBER, A.L., CREWS,
N.R., EZERSKIY, L.A., RENDELL, V.R., BELCHEVA, M.M., COSCIA, C.J.: Morphine modulation of
thrombospondin levels in astrocytes and its implications for neur ite outgrowth and synapse formation. J.
Biol. Chem., 285, 38415-38427 (2010)
IKEDA, H., KOTANI, A., KOSHIKAWA, N., COOLS, A.R.: Differential role of GABA A and GABA B
receptors in two distinct output stations of the rat striatum: Studies on the substantia n igra pars reticulate
and the globus pallidus. Neuroscience, 167, 31 -39 (2010)
IKEDA, H., KOTANI, A., LEE, J., KOSHIKAWA, N., COOLS, A.R.: GABA A receptors in the
mediodorsal thalamus play a crucial role in rat shell -specific acetylcholine-mediated, but not
dopamine-mediated, turning behaviour. Neuroscience, 159, 1200 -1207 (2009)
IKEDA, H., KOTANI, A., KOSHIKAWA, N., COOLS, A.R.: Somatostatin receptors in the
nucleus accumbens modulate dopamine-dependent but not acetylcholine-dependent turning
behaviour of rats. Neuroscience, 159, 974-981 (2009)
KOTANI, A., IKEDA, H., KOSHIKAWA, N., COOLS, A.R.: Role of orexin receptors in the
nucleus accumbens in dopamine-dependent turning behaviour of rats. Neuropharmacology,
54, 613-619 (2008)
IKEDA, H., KOTANI, A., KOSHIKAWA, N., COOLS, A.R.: A vehicle injection into the right
core of the nucleus accumbens both reverses the region-specificity and alters the type of
contralateral turning elicited by unilateral stimulation of dopamine D 2 /D 3 and D 1 receptors in
the left core of the nucleus accumbens. Eur. J. Pharmacol., 577, 64 -70 (2007)
IKEDA, H., MORIBE, S., SATO, M., KOTANI, A., KOSHIKAWA, N., COOLS, A.R.: Adrenergic
receptors in the nucleus accumbens shell differentially modulate dopamine and acetylcholine
receptor-mediated turning behaviour. Eur. J. Pharmacol., 554, 175 -182 (2007)
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